2,3-Dimercaptopropanol inhibits Ca2+ transport in microsomes from brain but not from fast-skeletal muscle.

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Effect of Mercuric Chloride Intoxication and Dimercaprol Treatment on σ‐Aminolevulinate Dehydratase from Brain, Liver and Kidney of Adult Mice

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Mechanisms involved in the cellular calcium homeostasis in vascular smooth muscle: Calcium pumps

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Are we ready to replace dimercaprol (BAL) as an arsenic antidote?

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Comparative Data of Ca2+ Transport in Brain and Skeletal Muscle Microsomes

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Characterisation of a novel Ca2+ pump inhibitor (bis-phenol) and its effects on intracellular Ca2+ mobilization

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Modifications of Cell Signalling in the Cytotoxicity of Metals

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The Ca2+-ATPase Isoforms of Platelets Are Located in Distinct Functional Ca2+ Pools and Are Uncoupled by a Mechanism Different from That of Skeletal Muscle Ca2+-ATPase

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Treatment of Mercuric Chloride Poisoning with Dimercaptosuccinic Acid and Diuretics: Preliminary Studies

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Alteration of Ca²⁺ Fluxes in Brain Microsomes by K⁺ and Na⁺: Modulation by Sulfated Polysaccharides and Trifluoperazine

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Investigations into the mechanism of 2,3-dimercaptopropanol neurotoxicity.

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BAL modulates glutamate transport in synaptosomes and synaptic vesicles from rat brain

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The SR Ca 2+ ATPase of the Antarctic scallop Adamussium colbecki : cold adaptation and heavy metal effects

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The importance of 2,3-dimercaptopropinol (British anti-lewisite, BAL) in the trypanocidal activity of topical melarsoprol

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Modifications of Ca2+ transport induced by glutathione in sarcoplasmic reticulum membranes of frog skeletal muscle.

2000 • 4 citations