Differential effects of 17[3‐estradiol on mitogen‐activated protein kinasepathways in rat cardiomyocytes

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How MAP Kinases Are Regulated

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MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo

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Parallel signal processing among mammalian MAPKs

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“Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the Myocardium

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Cardiac myocytes and fibroblasts contain functional estrogen receptors¹

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The coordinated action of protein tyrosine phosphatases and kinases in cell signaling

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A Role for the p38 Mitogen-activated Protein Kinase Pathway in Myocardial Cell Growth, Sarcomeric Organization, and Cardiac-specific Gene Expression

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Stimulation of the p38 Mitogen-activated Protein Kinase Pathway in Neonatal Rat Ventricular Myocytes by the G Protein–coupled Receptor Agonists, Endothelin-1 and Phenylephrine: A Role in Cardiac Myocyte Hypertrophy?

1998 • 313 citations

Relationship of Cardiovascular Risk Factors to Echocardiographic Left Ventricular Mass in Healthy Young Black and White Adult Men and Women

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Effect of 17β‐oestradiol on contraction, Ca²⁺ current and intracellular free Ca²⁺ in guinea‐pig isolated cardiac myocytes

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Rapid modulation of L‐type calcium current by acutely applied oestrogens in isolated cardiac myocytes from human, guinea‐pig and rat

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Differential effects of 17[3‐estradiol on mitogen‐activated protein kinasepathways in rat cardiomyocytes

1999 • 71 citations

Growth factor activation of the estrogen receptor in vascular cells occurs via a mitogen-activated protein kinase-independent pathway.

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Estrogen Stimulates Delayed Mitogen-Activated Protein Kinase Activity in Human Endothelial Cells via an Autocrine Loop That Involves Basic Fibroblast Growth Factor

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