Soluble Fas (APO‐1, CD95) and soluble Fas ligand in rheumatic diseases
Data up to Jan 2025
Total Citations Per Year
Abstract
References (14)
Derivation of the sledai. A disease activity index for lupus patients
1992 • 4,410 citations
Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis
1992 • 2,932 citations
Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor family
1993 • 2,546 citations
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation
1995 • 1,549 citations
Protection from Fas-Mediated Apoptosis by a Soluble Form of the Fas Molecule
1994 • 1,120 citations
Metalloproteinase-mediated release of human Fas ligand.
1995 • 835 citations
Fas ligand in human serum
1996 • 682 citations
Expression of the Fas ligand in cells of T cell lineage.
1995 • 659 citations
Expression of the functional soluble form of human fas ligand in activated lymphocytes.
1995 • 656 citations
Apoptotic signaling through CD95 (Fas/Apo-1) activates an acidic sphingomyelinase.
1994 • 582 citations
Accelerated in vitro apoptosis of lymphocytes from patients with systemic lupus erythematosus.
1994 • 555 citations
Levels of soluble fas/APO‐1 /CD95 in systemic lupus erythematosus and juvenile rheumatoid arthritis
1995 • 84 citations
Lack of correlation between serum soluble fas/APO‐1 levels and autoimmune disease
1995 • 63 citations
Rheumatoid arthritis treated with tenidap and piroxicam clinical associations with cytokine modulation by tenidap
1995 • 26 citations