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Lack of correlation between serum soluble fas/APO‐1 levels and autoimmune disease

Data up to Jan 2025

Published1995
Citations63
References15

Total Citations Per Year

Abstract

References (15)

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Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation

1995 • 1,549 citations

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1994 • 1,549 citations

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1995 • 1,346 citations

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1991 • 1,185 citations

Protection from Fas-Mediated Apoptosis by a Soluble Form of the Fas Molecule

1994 • 1,120 citations

Fas ligand mediates activation-induced cell death in human T lymphocytes.

1995 • 949 citations

The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

1994 • 708 citations

The mouse fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster

1994 • 335 citations

The apoptosis-1/Fas protein in human systemic lupus erythematosus.

1994 • 200 citations

Regulation of apoptosis and T cell activation by Fas-specific mAb

1994 • 107 citations

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Lack of correlation between serum soluble fas/APO‐1 levels and autoimmune disease (1995) – Arthritis & Rheumatism | Metascience Observatory Explorer