Altered chemokine levels in individuals at risk of Type 1 diabetes mellitus

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Autoantigens IA-2 and GAD in Type I (insulin-dependent) diabetes

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Differential Expression of CC Chemokines and the CCR5 Receptor in the Pancreas Is Associated with Progression to Type I Diabetes

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Only Multiple Autoantibodies to Islet Cells (ICA), Insulin, GAD65, IA-2 and IA-2β Predict Immune-Mediated (Type 1) Diabetes in Relatives

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Elevated C-Reactive Protein Levels in the Development of Type 1 Diabetes

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Optimized autoantibody-based risk assessment in family members. Implications for future intervention trials.

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Serum levels of the interferon-γ-inducing cytokine interleukin-18 are increased in individuals at high risk of developing Type I diabetes

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Characterization of Preparations of GAD65, Proinsulin, and the Islet Tyrosine Phosphatase IA-2 for Use in Detection of Autoreactive T-Cells in Type 1 Diabetes

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An Association of Autoantibody Status and Serum Cytokine Levels in Type 1 Diabetes

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Neither B Lymphocytes Nor Antibodies Directed Against Self Antigens of the Islets of Langerhans Are Required for Development of Virus-Induced Autoimmune Diabetes

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