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The mitochondrial toxin 3-nitropropionic acid induces vacuous chewing movements in rats. Implications for tardive dyskinesia?

Data up to Jan 2025

Published1995
Citations15
References16

Total Citations Per Year

Abstract

References (16)

Oxidative Stress, Glutamate, and Neurodegenerative Disorders

1993 • 3,746 citations

Oxidative damage and mitochondrial decay in aging.

1994 • 2,024 citations

Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

1993 • 1,017 citations

Duplication of biochemical changes of Huntington's chorea by intrastriatal injections of glutamic and kainic acids

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Neuroleptic medications inhibit complex I of the electron transport chain

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1994 • 203 citations

Tardive Dyskinesia

1993 • 187 citations

Haloperidol-induced morphological changes in striatum are associated with glutamate synapses

1994 • 135 citations

Schizophrenia, Affective Psychoses, and Other Disorders Treated with Neuroleptic Drugs: The Enigma of Tardive Dyskinesia, Its Neurobiological Determinants, and the Conflict of Paradigms

1989 • 99 citations

Systemic 3-nitropropionic acid: long-term effects on locomotor behavior

1994 • 62 citations

Study of neuropathologic changes in the striatum following 4, 8 and 12 months of treatment with fluphenazine in rats

1992 • 60 citations

GM1 ganglioside attenuates the development of vacuous chewing movements induced by long-term haloperidol treatment of rats

1994 • 35 citations

The action of chlorpromazine and imipramine on rat brain mitochondria

1979 • 26 citations

The effect of phenothiazine on the metabolism of liver mitochondria

1965 • 22 citations

The relationship between motor effects in rats following acute and chronic haloperidol treatment

1994 • 17 citations

Deleted Work

1955 • 0 citations

Cited By (0)

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The mitochondrial toxin 3-nitropropionic acid induces vacuous chewing movements in rats.… (1995) – Psychopharmacology | Metascience Observatory Explorer