Epidermal growth factor receptor is a cellular receptor for human cytomegalovirus

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The epidermal growth factor receptor family as a central element for cellular signal transduction and diversification.

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All ErbB Receptors Other Than the Epidermal Growth Factor Receptor Are Endocytosis Impaired

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Heterodimerization and functional interaction between EGF receptor family members: a new signaling paradigm with implications for breast cancer research

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Neu and its ligands: From an oncogene to neural factors

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Bivalence of EGF-like ligands drives the ErbB signaling network

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Human Cytomegalovirus Up-Regulates the Phosphatidylinositol 3-Kinase (PI3-K) Pathway: Inhibition of PI3-K Activity Inhibits Viral Replication and Virus-Induced Signaling

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Human cytomegalovirus penetrates host cells by PH-independent fusion at the cell surface

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Global modulation of cellular transcription by human cytomegalovirus is initiated by viral glycoprotein B

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The human cytomegalovirus UL55 (gB) and UL75 (gH) glycoprotein ligands initiate the rapid activation of Sp1 and NF-kappaB during infection

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Engagement of the Cellular Receptor for Glycoprotein B of Human Cytomegalovirus Activates the Interferon-Responsive Pathway

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Cytomegalovirus and atherosclerosis.

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Fiber Swap between Adenovirus Subgroups B and C Alters Intracellular Trafficking of Adenovirus Gene Transfer Vectors

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Pathogenic poxviruses reveal viral strategies to exploit the ErbB signaling network

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Activation of the Mitogen-Activated Protein Kinase p38 by Human Cytomegalovirus Infection through Two Distinct Pathways: a Novel Mechanism for Activation of p38

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Phosphatidylinositol 3′-Kinase-independent p70 S6 Kinase Activation by Fibroblast Growth Factor Receptor-1 Is Important for Proliferation but Not Differentiation of Endothelial Cells

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Molecular Aspects of Human Cytomegalovirus Diseases

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Direct interaction between human cytomegalovirus glycoprotein B and cellular annexin II

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Pertussis toxin-sensitive G proteins as mediators of the signal transduction pathways activated by cytomegalovirus infection of smooth muscle cells.

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Human cytomegalovirus infection inhibits epidermal growth factor (EGF) signalling by targeting EGF receptors

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EGF-Dependent Association of Phospholipase C-γ1 with c-Cbl

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