Retinal microenvironment controls resident and infiltrating macrophage function during uveoretinitis.

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Reprogramming of Lipopolysaccharide-Primed Macrophages Is Controlled by a Counterbalanced Production of IL-10 and IL-12

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Neutralizing TNF-alpha Activity Modulates T-cell Phenotype and Function in Experimental Autoimmune Uveoretinitis

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Macrophages in acute glomerular inflammation

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Endotoxin-induced uveitis in the rat is attenuated by inhibition of nitric oxide production.

1994 • 113 citations

Flow cytometric identification of a minority population of MHC class II positive cells in the normal rat retina distinct from CD45lowCD11b/c+CD4low parenchymal microglia.

1995 • 112 citations

Marrow-derived activated macrophages are required during the effector phase of experimental autoimmune uveoretinitis in rats

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1994 • 99 citations

Nitric oxide accelerates the onset and increases the severity of experimental autoimmune uveoretinitis through an IFN-gamma-dependent mechanism.

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Recruitment of antigen-nonspecific cells plays a pivotal role in the pathogenesis of a T cell-mediated organ-specific autoimmune uveoretinitis

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Immunomodulation of experimental autoimmune uveoretinitis: A model of tolerance induction with retinal antigens

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