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Fos is a Preferential Target of Glucocorticoid Receptor Inhibition of AP-1 Activity In Vitro

Data up to Jan 2025

Published1993
Citations15
References20

Total Citations Per Year

Abstract

References (20)

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

1983 • 11,902 citations

Antitumor promotion and antiinflammation: Down-modulation of AP-1 (Fos/Jun) activity by glucocorticoid hormone

1990 • 1,605 citations

Transcriptional interference between c-Jun and the glucocorticoid receptor: Mutual inhibition of DNA binding due to direct protein-protein interaction

1990 • 1,583 citations

Transcription Factor Interactions: Selectors of Positive or Negative Regulation from a Single DNA Element

1990 • 1,326 citations

Functional antagonism between oncoprotein c-Jun and the glucocorticoid receptor

1990 • 1,272 citations

Retinoic acid is a negative regulator of AP-1-responsive genes.

1991 • 542 citations

Jun-Fos and receptors for vitamins A and D recognize a common response element in the human osteocalcin gene

1990 • 522 citations

Activation of the ovalbumin gene by the estrogen receptor involves the Fos-Jun complex

1990 • 486 citations

Negative regulation of the rat stromelysin gene promoter by retinoic acid is mediated by an AP1 binding site.

1990 • 377 citations

Fos-Jun heterodimers and jun homodimers bend DNA in opposite orientations: Implications for transcription factor cooperativity

1991 • 296 citations

Mutual transrepression of Fos and the glucocorticoid receptor: involvement of a functional domain in Fos which is absent in FosB.

1990 • 290 citations

Interference between pathway-specific transcription factors: glucocorticoids antagonize phorbol ester-induced AP-1 activity without altering AP-1 site occupation in vivo.

1992 • 282 citations

Characterization of a Monoclonal Antibody to the Rat Liver Glucocorticoid Receptor*

1984 • 263 citations

Coordinate occupancy of AP-1 sites in the vitamin D-responsive and CCAAT box elements by Fos-Jun in the osteocalcin gene: model for phenotype suppression of transcription.

1990 • 209 citations

Cell-specific inhibitory and stimulatory effects of Fos and Jun on transcription activation by nuclear receptors.

1991 • 195 citations

Altered protein conformation on DNA binding by Fos and Jun

1990 • 193 citations

DNA Bending by Fos and Jun: The Flexible Hinge Model

1991 • 186 citations

Co-localization of elements required for phorbol ester stimulation and glucocorticoid repression of proliferin gene expression.

1989 • 154 citations

The basic region of Fos mediates specific DNA binding.

1989 • 132 citations

Transcriptional stimulation by CaPO4-DNA precipitates

1988 • 49 citations

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Fos is a Preferential Target of Glucocorticoid Receptor Inhibition of AP-1 Activity In… (1993) – Molecular and Cellular Biology | Metascience Observatory Explorer