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ENDOTHELIAL CELL ACTIVATION AND HIGH INTERLEUKIN-1 SECRETION IN THE PATHOGENESIS OF ACUTE KAWASAKI DISEASE

Data up to Jan 2025

Published1989
Citations373
References31
Clinical Trials (1)

Total Citations Per Year

Abstract

References (31)

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1984 • 485 citations

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1986 • 378 citations

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1986 • 314 citations

Two monokines, interleukin 1 and tumor necrosis factor, render cultured vascular endothelial cells susceptible to lysis by antibodies circulating during Kawasaki syndrome.

1986 • 275 citations

Immunoglobulin M antibodies present in the acute phase of Kawasaki syndrome lyse cultured vascular endothelial cells stimulated by gamma interferon.

1986 • 244 citations

Molecular cloning and expression of human tumor necrosis factor and comparison with mouse tumor necrosis factor

1985 • 240 citations

Endothelial activation during interleukin 2 immunotherapy. A possible mechanism for the vascular leak syndrome.

1988 • 225 citations

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1986 • 220 citations

Peripheral blood monocyte/macrophages and serum tumor necrosis factor in Kawasaki disease

1988 • 207 citations

Reversal of lymphocyte activation in vivo in the Kawasaki syndrome by intravenous gammaglobulin.

1987 • 179 citations

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1987 • 171 citations

Expression and characterization of the product of a human immune interferon cDNA gene in Chinese hamster ovary cells.

1983 • 167 citations

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1986 • 154 citations

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1982 • 125 citations

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1983 • 105 citations

High-dose gammaglobulin therapy for Kawasaki disease

1987 • 101 citations

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1987 • 54 citations

SERUM INTERFERON CONCENTRATIONS AND RETROVIRAL SEROLOGY IN KAWASAKI SYNDROME

1988 • 37 citations

Serum interferon concentrations and retroviral serology in Kawasaki syndrome.

1988 • 32 citations

Intravenous γ-Globulin Use in Children With Kawasaki Disease

1988 • 17 citations

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ENDOTHELIAL CELL ACTIVATION AND HIGH INTERLEUKIN-1 SECRETION IN THE PATHOGENESIS OF ACUTE… (1989) – The Lancet | Metascience Observatory Explorer