Back to search

c-Jun is essential for normal mouse development and hepatogenesis

Data up to Jan 2025

Published1993
Citations543
References24

Total Citations Per Year

Abstract

References (24)

A Rapid and Sensitive Method for the Quantitation of Microgram Quantities of Protein Utilizing the Principle of Protein-Dye Binding

1976 • 225,752 citations

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

1976 • 200,127 citations

The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation

1991 • 3,396 citations

Effects of an Rb mutation in the mouse

1992 • 1,779 citations

Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis

1992 • 1,285 citations

Requirement for a functional Rb-1 gene in murine development

1992 • 1,015 citations

Bone and haematopoietic defects in mice lacking c-fos

1992 • 939 citations

Teratocarcinomas and embryonic stem cells : a practical approach

1987 • 878 citations

Pleiotropic effects of a null mutation in the c-fos proto-oncogene

1992 • 643 citations

Characterization of junD: a new member of the jun proto-oncogene family.

1989 • 492 citations

Mice homozygous for a targeted disruption of the proto-oncogeneint-2have developmental defects in the tail and inner ear

1993 • 446 citations

Avian sarcoma virus 17 carries the jun oncogene.

1987 • 445 citations

The jun and fos protein families are both required for cell cycle progression in fibroblasts.

1991 • 442 citations

Functional antagonism between c-Jun and MyoD proteins: A direct physical association

1992 • 424 citations

Osteoblasts are target cells for transformation in c-fos transgenic mice

1993 • 347 citations

Teratocarcinomas and Embryonic Stem Cells: A Practical Approach

1988 • 263 citations

Tissue-specific expression of c-jun and junB during organogenesis in the mouse

1989 • 172 citations

A novel target cell for c-fos-induced oncogenesis: development of chondrogenic tumours in embryonic stem cell chimeras.

1991 • 172 citations

Overexpression of c-jun, junB, or junD affects cell growth differently.

1991 • 136 citations

Ectopic expression of c-jun leads to differentiation of P19 embryonal carcinoma cells.

1990 • 111 citations

Embryonic stem (ES) cells lacking functional c-jun: consequences for growth and differentiation, AP-1 activity and tumorigenicity.

1992 • 74 citations

A potential role for c-jun in cell cycle progression through late G1 and S.

1991 • 55 citations

High molecular weight component of Mallory bodies detected by a monoclonal antibody.

1990 • 39 citations

Transcriptional regulation in the testis: a role for transcription factor AP-1 complexes at various stages of spermatogenesis.

1993 • 37 citations

Cited By (0)

Loading...
c-Jun is essential for normal mouse development and hepatogenesis (1993) – Nature | Metascience Observatory Explorer