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Evidence that DNA Damage Is a Mediate in Ultraviolet B Radiation-Induced Inhibition of Human Gene Expression: Ultraviolet B Radiation Effects on Intercellular Adhesion Molecule-1 (ICAM-1) Expression

Data up to Jan 2025

Published1994
Citations45
References22

Total Citations Per Year

Abstract

References (22)

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Primary structure of ICAM-1 demonstrates interaction between members of the immunoglobulin and integrin supergene families

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Adhesion of T lymphoblasts to epidermal keratinocytes is regulated by interferon gamma and is mediated by intercellular adhesion molecule 1 (ICAM-1).

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Ultraviolet Radiation Can Either Suppress or Induce Expression of Intercellular Adhesion Molecule 1 (ICAM-1) on the Surface of Cultured Human Keratinocytes

1990 • 137 citations

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Tumor Necrosis Factor β and Ultraviolet Radiation Are Potent Regulators of Human Keratinocyte ICAM-1 Expression

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The 55-kD tumor necrosis factor receptor on human keratinocytes is regulated by tumor necrosis factor-alpha and by ultraviolet B radiation.

1993 • 93 citations

Ultraviolet Radiation Effects on Human Keratinocyte ICAM-1 Expression: UV-Induced Inhibition of Cytokine-Induced ICAM-1 mRNA Expression Is Transient, Differentially Restored for IFNγ Versus TNFα, and Followed by ICAM-1 Induction Via a TNFα-Like Pathway

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Systemic suppression of delayed-type hypersensitivity by supernatants from UV-irradiated keratinocytes

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Modulation of the expression of intercellular adhesion molecule-1 (ICAM-1) in human keratinocytes by ultraviolet (UV) radiation

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Evidence that DNA Damage Is a Mediate in Ultraviolet B Radiation-Induced Inhibition of… (1994) – Journal of Investigative Dermatology | Metascience Observatory Explorer