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Duration of Glutamate Release After Acute Ischemic Stroke

Data up to Jan 2025

Published1997
Citations179
References15

Total Citations Per Year

Abstract

References (15)

Excitatory Amino Acids as a Final Common Pathway for Neurologic Disorders

1994 • 2,668 citations

Glutamate and the pathophysiology of hypoxic–ischemic brain damage

1986 • 2,428 citations

Glutamate neurotoxicity in cortical cell culture

1987 • 1,400 citations

Clinical Experience With Excitatory Amino Acid Antagonist Drugs

1995 • 499 citations

Progression of ischaemic stroke and excitotoxic aminoacids

1997 • 324 citations

Neuroexcitatory Amino Acids and Their Relation to Infarct Size and Neurological Deficit in Ischemic Stroke

1996 • 252 citations

Progressing Neurological Deficit Secondary to Acute Ischemic Stroke

1995 • 236 citations

Deteriorating ischemic stroke

1990 • 229 citations

Changes in Amino Acid Neurotransmitters and Cerebral Blood Flow in the Ischemic Penumbral Region following Middle Cerebral Artery Occlusion in the Rat: Correlation with Histopathology

1993 • 220 citations

Excitatory amino acids in cerebrospinal fluid following traumatic brain injury in humans

1993 • 208 citations

Massive Persistent Release of Excitatory Amino Acids Following Human Occlusive Stroke

1995 • 151 citations

Elevation of Neuroactive Substances in the Cortex of Cats during Prolonged Focal Ischemia

1993 • 102 citations

Intraischemic Hypothermia Decreases the Release of Glutamate in the Cores of Permanent Focal Cerebral Infarcts

1995 • 99 citations

Effect of P2Y Agonists on Adenosine Transport in Cultured Chromaffin Cells

1993 • 42 citations

The Rationale for Glutamate Antagonists in the Treatment of Traumatic Brain Injury

1995 • 34 citations

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Duration of Glutamate Release After Acute Ischemic Stroke (1997) – Stroke | Metascience Observatory Explorer