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Role of nitric oxide in long-term angiotensin II-induced renal vasoconstriction.

Data up to Jan 2025

Published1993
Citations26
References20

Total Citations Per Year

Abstract

References (20)

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.

1987 • 5,027 citations

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Chronic blockade of nitric oxide synthesis in the rat produces systemic hypertension and glomerular damage.

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1988 • 90 citations

Regulation of glomerular filtration rate and sodium excretion by angiotensin II.

1986 • 78 citations

NG-monomethyl-L-arginine and nitroarginine potentiate pressor responsiveness of vasoconstrictors in conscious rats

1992 • 61 citations

Pressor and renal vasoconstrictor effects of NG-nitro-L-arginine as affected by blockade of pressor mechanisms mediated by the sympathetic nervous system, angiotensin, prostanoids and vasopressin.

1992 • 60 citations

Angiotensin II: Preferential Efferent Constriction?

1986 • 32 citations

Endothelium‐dependent modulation of the pressor activity of arginine vasopressin in the isolated superior mesenteric arterial bed of the rat

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Effects of hypoproteinemia on renal hemodynamics, arterial pressure, and fluid volume

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Regulation of cardiac output during aldosterone-induced hypertension

1989 • 18 citations

Protection of preglomerular vessels from angiotensin II vasoconstriction by renal prostaglandins.

1985 • 18 citations

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Role of nitric oxide in long-term angiotensin II-induced renal vasoconstriction. (1993) – Hypertension | Metascience Observatory Explorer