The requirement for pertussis to induce EAU is strain-dependent: B10.RIII, but not B10.A mice, develop EAU and Th1 responses to IRBP without pertussis treatment.

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A new model of autoimmune disease. Experimental autoimmune uveoretinitis induced in mice with two different retinal antigens.

1988 • 451 citations

Monocyte chemotactic proteins MCP‐1, MCP‐2, and MCP‐3 are major attractants for human CD4 ⁺ and CD8 ⁺ T lymphocytes

1994 • 322 citations

Strength of TCR signal determines the costimulatory requirements for Th1 and Th2 CD4+ T cell differentiation.

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IFN-gamma-deficient mice develop experimental autoimmune uveitis in the context of a deviant effector response.

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Pertussis toxin inhibits migration of B and T lymphocytes into splenic white pulp cords.

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Genetic susceptibility to experimental autoimmune uveitis involves more than a predisposition to generate a T helper-1-like or a T helper-2-like response.

1997 • 133 citations

Genetic susceptibility to experimental autoimmune uveoretinitis in the rat is associated with an elevated Th1 response.

1996 • 129 citations

Endogenous IL-12 Is Required for Induction and Expression of Experimental Autoimmune Uveitis

1998 • 110 citations

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Site-selective homing of antigen-primed lymphocyte populations can play a crucial role in the efferent limb of cell-mediated immune responses in vivo.

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Pertussis toxin pretreatment alters the in vivo cell division behaviour and survival of B lymphocytes after intravenous transfer

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Pertussigen enhances antigen-driven interferon-γ production by sensitized lymphoid cells

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The effects of pertussis toxin on the induction and transfer of experimental autoimmune uveoretinitis

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Pertussigen in vivo enhances antigen-specific production in vitro of lymphokine that stimulates macrophage procoagulant activity and plasminogen activator.

1986 • 10 citations