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Short-lived complexes between myelin basic protein peptides and IAk.

Data up to Jan 2025

Published1994
Citations24
References16

Total Citations Per Year

Abstract

References (16)

Binding of immunogenic peptides to Ia histocompatibility molecules

1985 • 1,209 citations

Isolation and characterization of antigen-la complexes involved in T cell recognition

1986 • 470 citations

Antigen recognition in autoimmune encephalomyelitis and the potential for peptide-mediated immunotherapy

1989 • 394 citations

The human class II MHC protein HLA-DR1 assembles as empty αβ heterodimers in the absence of antigenic peptide

1992 • 306 citations

Antigen presentation by supported planar membranes containing affinity-purified I-Ad.

1984 • 281 citations

An autoantigenic T cell epitope forms unstable complexes with class II MHC: a novel route for escape from tolerance induction

1993 • 182 citations

High-affinity binding of an influenza hemagglutinin-derived peptide to purified HLA-DR.

1990 • 148 citations

MHC class II function preserved by low-affinity peptide interactions preceding stable binding

1994 • 135 citations

T cell receptor antagonist peptides are highly effective inhibitors of experimental allergic encephalomyelitis

1994 • 123 citations

A kinetic intermediate in the reaction of an antigenic peptide and I–Ek

1989 • 122 citations

Kinetic intermediates in the reactions between peptides and proteins of major histocompatibility complex class II.

1994 • 47 citations

High affinity for class II molecules as a necessary but not sufficient characteristic of encephalitogenic determinants

1992 • 39 citations

Enhanced immunogenicity of a T cell immunogenic peptide by modifications of its N and C termini

1989 • 29 citations

A first-order reaction controls the binding of antigenic peptides to major histocompatibility complex class II molecules.

1991 • 29 citations

Inhibition of experimental autoimmune encephalomyelitis by MHC class II binding competitor peptides depends on the relative MHC binding affinity of the disease-inducing peptide.

1994 • 25 citations

Polymorphic residues on the I-A beta chain modulate the stimulation of T cell clones specific for the N-terminal peptide of the autoantigen myelin basic protein.

1989 • 18 citations

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Short-lived complexes between myelin basic protein peptides and IAk. (1994) – Proceedings of the National Academy of Sciences | Metascience Observatory Explorer