Model of robust induction of glomerulosclerosis in mice: Importance of genetic background

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Method of renal mass reduction is a critical modulator of subsequent hypertension and glomerular injury.

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Functional and Structural Correlates of Glomerulosclerosis after Renal Mass Reduction in the Rat

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Strain differences rather than hyperglycemia determine the severity of glomerulosclerosis in mice

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Dissociation of glomerular hypertrophy, cell proliferation, and glomerulosclerosis in mouse strains heterozygous for a mutation (Os) which induces a 50% reduction in nephron number.

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Analysis of renal structural and functional features in two rat strains with a different susceptibility to glomerular sclerosis.

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In Silico Quantitative Trait Locus Map for Atherosclerosis Susceptibility in Apolipoprotein E–Deficient Mice

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Nature and Severity of the Glomerular Response to Nephron Reduction Is Strain-Dependent in Mice

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Distinction in Genetic Determinants for Injury-Induced Neointimal Hyperplasia and Diet-Induced Atherosclerosis in Inbred Mice

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Role of angiotensin II in glomerular injury

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Increased genetic susceptibility to renal damage in the stroke-prone spontaneously hypertensive rat

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Genetic Regulation of the Development of Glomerular Sclerotic Lesions in the BUF/Mna Rat

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