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Aggressive Fibromatosis (Desmoid Tumor) is A Monoclonal Disorder

Data up to Jan 2025

Published1997
Citations138
References14

Total Citations Per Year

Abstract

References (14)

Methylation of HpaII and HhaI sites near the polymorphic CAG repeat in the human androgen-receptor gene correlates with X chromosome inactivation.

1992 • 1,686 citations

Hereditary cancer, oncogenes, and antioncogenes.

1985 • 1,261 citations

Langerhans'-Cell Histiocytosis (Histiocytosis X) -- A Clonal Proliferative Disease

1994 • 999 citations

Clonal analysis using recombinant DNA probes from the X-chromosome.

1987 • 439 citations

PCR bias in amplification of androgen receptor alleles, a trinucleotide repeat marker used in clonality studies

1995 • 193 citations

Coexistence of somatic and germ-line mutations of APC gene in desmoid tumors from patients with familial adenomatous polyposis.

1993 • 152 citations

Clonal chromosomal abnormalities in desmoid tumors. Implications for histopathogenesis

1992 • 151 citations

A polymerase chain reaction assay for non-random X chromosome inactivation identifies monoclonal endometrial cancers and precancers.

1995 • 106 citations

Activating mutations of Gs protein in monostotic fibrous lesions of bone

1996 • 89 citations

Novel mutations and inactivation of both alleles of the APC gene in desmoid tumors

1995 • 62 citations

Platelet‐derived growth factor in fibrous musculoskeletal disorders: A study of pathologic tissue sections and in vitro primary cell cultures

1995 • 57 citations

Some desmoid tumors are characterized by trisomy 8

1994 • 50 citations

Aggressive Fibromatosis

1992 • 43 citations

Molecular Genetic and Immunohistochemical Analysis of the Tumor Suppressor Genes Rb and p53 in Palmar and Aggressive Fibromatosis

1996 • 40 citations

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Aggressive Fibromatosis (Desmoid Tumor) is A Monoclonal Disorder (1997) – Diagnostic Molecular Pathology | Metascience Observatory Explorer