GABAB receptor activation inhibits 5-hydroxytryptamine-stimulated inositol phospholipid turnover in mouse cerebral cortex
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References (9)
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5‐Hydroxytryptamine‐Stimulated Inositol Phospholipid Hydrolysis in the Mouse Cortex Has Pharmacological Characteristics Compatible with Mediation via 5‐HT2 Receptors but This Response Does Not Reflect Altered 5‐HT2 Function After 5,7‐Dihydroxytryptamine Lesioning or Repeated Antidepressant Treatments
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The effects of the gaba-mimetic drugs, progabide and baclofen, on the biochemistry and function of 5-hydroxytryptamine and noradrenaline
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