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17q gain in neuroblastoma predicts adverse clinical outcome

Data up to Jan 2025

Published2001
Citations71
References16
Clinical Trials (1)

Total Citations Per Year

Abstract

References (16)

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Gain of chromosome 17 is the most frequent abnormality detected in neuroblastoma by comparative genomic hybridization.

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Allelic loss of chromosome 1 and additional chromosome 17 material are both unfavourable prognostic markers in neuroblastoma

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Genetic heterogeneity of neuroblastoma studied by comparative genomic hybridization

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Gain of chromosome arm 17q predicts unfavourable outcome in neuroblastoma patients

1997 • 111 citations

Translocation involving 1p and 17q is a recurrent genetic alteration of human neuroblastoma cells.

1994 • 111 citations

COMPARATIVE GENOMIC HYBRIDIZATION (CGH) ANALYSIS OF NEUROBLASTOMAS—AN IMPORTANT METHODOLOGICAL APPROACH IN PAEDIATRIC TUMOUR PATHOLOGY

1997 • 106 citations

Additional copies of a 25 Mb chromosomal region originating from 17q23.1-17qter are present in 90% of high-grade neuroblastomas

1996 • 100 citations

Comparative genomic hybridization study of primary neuroblastoma tumors. United Kingdom Children's Cancer Study Group.

1997 • 93 citations

Comparative genomic hybridization study of primary neuroblastoma tumors

1997 • 73 citations

Promiscuous translocations of chromosome arm 17q in human neuroblastomas

1997 • 62 citations

Molecular cytogenetic delineation of 17q translocation breakpoints in neuroblastoma cell lines

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17q gain in neuroblastoma predicts adverse clinical outcome (2001) – Medical and Pediatric Oncology | Metascience Observatory Explorer