Asymptomatic Helicobacter pylori gastritis is associated with increased sucrose permeability.

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A simple, non-invasive marker of gastric damage: sucrose permeability

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Pathophysiology of Helicobacter pylori infection.

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The Movement of Solutes and Cells across Tight Junctionsa

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Positive Correlation between H,K-Adenosine Triphosphatase Autoantibodies and Helicobacter pylori Antibodies in Patients with Pernicious Anemia

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IgA antiendomysium antibodies have a high positive predictive value for celiac disease in asymptomatic patients

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Neutrophil-derived oxidants mediate formyl-methionyl-leucyl-phenylalanine-induced increases in mucosal permeability in rats

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Dyspeptic Symptoms and Gastric Emptying of Solids in Patients with Functional Dyspepsia: Role ofHelicobacter pyloriInfection

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High incidence of Helicobacter pylori colonization in early gastric cancer and the possible relationship to carcinogenesis

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Evaluation of differential disaccharide excretion in urine for non-invasive investigation of altered intestinal disaccharidase activity caused by alpha-glucosidase inhibition, primary hypolactasia, and coeliac disease.

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Increased Gastric Absorption of Polyethylene Glycols in Atrophic Gastritis

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